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Model of ferroptosis and key molecules in the adrenal cortex. ACSL4... |  Download Scientific Diagram
Model of ferroptosis and key molecules in the adrenal cortex. ACSL4... | Download Scientific Diagram

ACSL4 dictates ferroptosis sensitivity by shaping cellular lipid  composition. - Abstract - Europe PMC
ACSL4 dictates ferroptosis sensitivity by shaping cellular lipid composition. - Abstract - Europe PMC

The positive feedback between ACSL4 expression and O-GlcNAcylation  contributes to the growth and survival of hepatocellular carcinoma | Aging
The positive feedback between ACSL4 expression and O-GlcNAcylation contributes to the growth and survival of hepatocellular carcinoma | Aging

Schematic model of the signaling pathway involved in ACSL4-induced  up-regulation of intramitochondrial AA, LOX products, COX-2 and PGE2  production in the development of an aggressive phenotype in breast cancer  cells.
Schematic model of the signaling pathway involved in ACSL4-induced up-regulation of intramitochondrial AA, LOX products, COX-2 and PGE2 production in the development of an aggressive phenotype in breast cancer cells.

ACSL4 Antibody (F-4) | SCBT - Santa Cruz Biotechnology
ACSL4 Antibody (F-4) | SCBT - Santa Cruz Biotechnology

Activation of ALOX12 by a multi-organelle-orienting photosensitizer drives  ACSL4-independent cell ferroptosis | Cell Death & Disease
Activation of ALOX12 by a multi-organelle-orienting photosensitizer drives ACSL4-independent cell ferroptosis | Cell Death & Disease

Functional Interaction between Acyl-CoA Synthetase 4, Lipooxygenases and  Cyclooxygenase-2 in the Aggressive Phenotype of Breast Cancer Cells | PLOS  ONE
Functional Interaction between Acyl-CoA Synthetase 4, Lipooxygenases and Cyclooxygenase-2 in the Aggressive Phenotype of Breast Cancer Cells | PLOS ONE

Positive feedback amplifies ferroptosis | Nature Cell Biology
Positive feedback amplifies ferroptosis | Nature Cell Biology

Frontiers | ACSL4 as a Potential Target and Biomarker for Anticancer: From  Molecular Mechanisms to Clinical Therapeutics
Frontiers | ACSL4 as a Potential Target and Biomarker for Anticancer: From Molecular Mechanisms to Clinical Therapeutics

Frontiers | ACSL4 as a Potential Target and Biomarker for Anticancer: From  Molecular Mechanisms to Clinical Therapeutics
Frontiers | ACSL4 as a Potential Target and Biomarker for Anticancer: From Molecular Mechanisms to Clinical Therapeutics

The primary mechanism of ferroptosis. ACSL4 acylates PUFA, which is... |  Download Scientific Diagram
The primary mechanism of ferroptosis. ACSL4 acylates PUFA, which is... | Download Scientific Diagram

Cancers | Free Full-Text | ACSL3 and ACSL4, Distinct Roles in Ferroptosis  and Cancers
Cancers | Free Full-Text | ACSL3 and ACSL4, Distinct Roles in Ferroptosis and Cancers

Acyl-CoA synthetase-4 is implicated in drug resistance in breast cancer  cell lines involving the regulation of energy-dependent transporter  expression - ScienceDirect
Acyl-CoA synthetase-4 is implicated in drug resistance in breast cancer cell lines involving the regulation of energy-dependent transporter expression - ScienceDirect

Schematic model of the signaling pathway involved in ACSL4-induced... |  Download Scientific Diagram
Schematic model of the signaling pathway involved in ACSL4-induced... | Download Scientific Diagram

Biology | Free Full-Text | The ACSL4 Network Regulates Cell Death and  Autophagy in Diseases
Biology | Free Full-Text | The ACSL4 Network Regulates Cell Death and Autophagy in Diseases

Identification of p115 as a novel ACSL4 interacting protein and its role in  regulating ACSL4 degradation - ScienceDirect
Identification of p115 as a novel ACSL4 interacting protein and its role in regulating ACSL4 degradation - ScienceDirect

Oleanolic acid inhibits cervical cancer Hela cell proliferation thro
Oleanolic acid inhibits cervical cancer Hela cell proliferation thro

ACSL4 deficiency confers protection against ferroptosis-mediated acute  kidney injury - ScienceDirect
ACSL4 deficiency confers protection against ferroptosis-mediated acute kidney injury - ScienceDirect

AIM2 promotes renal cell carcinoma progression and sunitinib resistance  through FOXO3a-ACSL4 axis-regulated ferroptosis [Abstract]
AIM2 promotes renal cell carcinoma progression and sunitinib resistance through FOXO3a-ACSL4 axis-regulated ferroptosis [Abstract]

Seco-Lupane Triterpene Derivatives Induce Ferroptosis through GPX4/ACSL4  Axis and Target Cyclin D1 to Block the Cell Cycle | Journal of Medicinal  Chemistry
Seco-Lupane Triterpene Derivatives Induce Ferroptosis through GPX4/ACSL4 Axis and Target Cyclin D1 to Block the Cell Cycle | Journal of Medicinal Chemistry

IJMS | Free Full-Text | ACSL4-Mediated Ferroptosis and Its Potential Role  in Central Nervous System Diseases and Injuries
IJMS | Free Full-Text | ACSL4-Mediated Ferroptosis and Its Potential Role in Central Nervous System Diseases and Injuries

Fatty acid oxidation protects cancer cells from apoptosis by increasing  mitochondrial membrane lipids
Fatty acid oxidation protects cancer cells from apoptosis by increasing mitochondrial membrane lipids

Seco-Lupane Triterpene Derivatives Induce Ferroptosis through GPX4/ACSL4  Axis and Target Cyclin D1 to Block the Cell Cycle | Journal of Medicinal  Chemistry
Seco-Lupane Triterpene Derivatives Induce Ferroptosis through GPX4/ACSL4 Axis and Target Cyclin D1 to Block the Cell Cycle | Journal of Medicinal Chemistry

Mechanism of ferroptosis. ACSL4 and LPCAT3 enzymes are required for the...  | Download Scientific Diagram
Mechanism of ferroptosis. ACSL4 and LPCAT3 enzymes are required for the... | Download Scientific Diagram

JCI - RB1-deficient prostate tumor growth and metastasis are vulnerable to  ferroptosis induction via the E2F/ACSL4 axis
JCI - RB1-deficient prostate tumor growth and metastasis are vulnerable to ferroptosis induction via the E2F/ACSL4 axis

ACSL4 Antibody (A-5) | SCBT - Santa Cruz Biotechnology
ACSL4 Antibody (A-5) | SCBT - Santa Cruz Biotechnology

ACSL4 suppresses glioma cells proliferation via activating ferroptosis
ACSL4 suppresses glioma cells proliferation via activating ferroptosis